Nce qe nkaug - dab tsi nws yuav hais tau lus?

Tsis yog txhua txhua ntawm peb muaj peev xwm to taub txog cov kev kho mob cov lus. Piv txwv li, yog tias kev kuaj tau qhia kom qe, nkaug, yog dab tsi nws yuav hais tau lus? Ua ntej, peb saib yog dab tsi lub sij hawm meant.

Yuav ua li cas yog lub qe?

Lwm lub npe - neutrophilic granulocytes. Nws yog ib yam ntawm cov ntshav dawb uas yog rau lub cev ntawm ib tug neeg tseem ceeb heev. Nws yog los ntawm cov ntshav dawb hauv lub cev yuav tiv taus cov kab mob kab mob. Qhov chaw ntawm tsim ntawm qe yog cov hlwb pob txha. Nyob rau hauv cov ntaub so ntswg ntawm lub cev lawv tuaj los ntawm cov ntshav thiab pab txhawb kom txoj kev puas tsuaj ntawm ib tug neeg txawv pathogens. Ces qe tuag.

Yog hais tias lub cev tau txais tej kab mob thiab tsim o, cov ntshav yog ntiab tawm band qe.

Nce qe nkaug - uas yuav ua rau xws li ib tug tshwm sim?

Nyob rau hauv cov neeg laus noj qab nyob zoo neeg cov ntshav cell theem ntawm 6% ntawm tag nrho cov leukocytes. Nyob rau hauv cov me nyuam, qhov no daim duab tej zaum yuav txawv nyob ntawm seb muaj hnub nyoog. Piv txwv li, rau ib tug me nyuam mos cai yog cov ntsiab lus ntawm band qe nyob rau hauv tus nqi ntawm 17%. Ua ntej lub xyoo no daim duab yuav tsum tsis kam mus rau 4%. Los ntawm ib lub xyoo los mus tiav hluas Neutrophil count nce mus rau 5%. Yog hais tias tus qhab qe yog nce nyob rau hauv cov me nyuam, cov ua ntawm no phenomenon yog tib yam li nyob rau hauv cov neeg laus.

Ua tus xov tooj qe nyob rau hauv cov ntshav yog hu ua neutrophilia.

Cov nab npawb ntawm cov qe nyob rau hauv cov ntshav qab vim li nram qab no yog vim li cas:

• mob inflammatory dab (mob ntsws, peritonitis, sepsis, otitis, hnyuv tws);
• intoxication (mercury, taug txhuas);
• endogenous intoxication nyob rau tom qab ntawm necrosis ntawm hepatocytes, mob ntshav qab zib, uremia, eclampsia;
• feem ntau nkaug qe yog tsa nyob rau hauv cev xeeb tub ;
• kab dab tshwm sim los ntawm cov kab mob, ib txhia kab mob, fungi, rickettsia, spirochetes;
• physiological thiab kev nyuaj siab.

Tshwj xeeb tshaj yog cov lus hais ntau zog nkaug qe nyob rau hauv cov ntshav nyob rau hauv purulent kab mob xws li ua paug thiab cellulitis.

Nyob rau hauv tsawg tus neeg mob, nce nyob rau hauv lub xov tooj ntawm cov ntshav yog lub txiaj ntsim ntawm myocardial infarction, bronchial hlav, mob plab, pancreas, mob stroke, venous ulcers, uas tau txais cov tshuaj (corticosteroids), uas nws kim heev kev kub nyhiab.

Ua rau tsis muaj ntshav qe

Qhov tsom xam yuav qhia tsis tau tsuas yog siab dua nkaug qe nyob rau hauv cov ntshav. Cov theem ntawm cov ntshav yuav raug muab txo. Qhov no tshwm sim yog hu ua "neutropenia". Ntxias yuav txo tau nyob rau hauv lub xov tooj qe nram qab no hais tias:

• kab mob (paratyphoid, typhoid kub taub hau, brucellosis);
• kab mob xws li tus kab mob tus kab mob siab, mob khaub thuas, ua qoob, rubella;
• hyperthyroidism;
• cov kab mob ntawm lub plawv system: hlau tsis muaj peev xwm, hypoplastic, aplastic, megaloblastic anemia, leukemia mob daim ntawv;
• anaphylactic schok;
• noj tej yam tshuaj (cytostatics, analgesics, potivosudorozhnyh txhais tau tias);
• ib tug kev tshuaj ntsuam genetic predisposition mus rau qhov mob.

Tam sim no uas koj paub tias dab tsi ua rau lub siab ntxiv thiab lub caij nplooj zeeg ntawm qe nyob rau hauv cov ntshav, raws li zoo raws li lawv permissible txwv, thiab yuav yooj yim txiav txim hauv lub xeev ntawm koj lub cev thaum lub caij.