Gout - dab tsi yog tus kab mob no thiab yuav ua li cas nws qhia nws tus kheej?

Kev ua txhaum ntawm uric acid metabolism thiab deposition ntawm nws cov ntsev nyob rau hauv cov pob qij txha yuav ua tau kom ib tug mob xws li gout. Yuav ua li cas yog tus kab mob no? Qhov no daim ntawv ntawm kev mob caj dab yus ob leeg kab mob thiab nrog vim metabolic mob.

Gout Kab Mob tej yam tshwm sim

Kab Mob manifested los ntawm mob heev, o thiab liab ntawm cov pob qij txha. Gout nres yuav nyob ntev li ntawm ob peb hnub mus rau ob peb lub lis piam. Nws feem ntau tshwm sim thaum hmo ntuj, tus txha yuav rhiab heev thiab kub mus rau lub kov. ua npaws tshwm sim ntau zaus. Yeej, tus kab mob no muaj nyob rau hauv cov pob qij txha ntawm lub toes, tab sis kuj muaj peev xwm cuam tshuam los ntawm tus ntiv tes, dab teg, hauv caug, thiab pob luj taws pob qij txha.

ntaub ntawv ntawm tus kab mob

thawj gout

Yuav ua li cas yog tus kab mob no? Qhov no yog ib tug mob nyob rau hauv uas ua txhaum yuav tsis muaj feem rau tej yam kab mob, thiab tus kab mob no muaj nws tus kheej.

theem nrab gout

Xws li ib tug mob yuav ua rau muaj mob lwm yam kab mob: kab mob plawv, psoriasis, hemoglobinopathies, myeloid leukemia thiab lwm yam kab mob ntawm cov ntshav thiab hauv nruab nrog cev.

Gout - dab tsi yog tus kab mob no thiab yog dab tsi nws ua?

Cov tsos ntawm cov kab mob no yog tshwm sim los ntawm ib tug kev ncua nyob rau hauv lub cev ntawm uric acid uas yog tsim raws li ib tug tshwm sim ntawm kev ntawm cov khoom uas muaj purines. Nyob rau hauv lub qub xeev ntawm uric acid yuav tsum tau tso tawm nyob rau hauv cov zis. Tab sis ib tug neeg txom nyem los ntawm gout, lub substance accumulates thiab yog tso rau hauv daim ntawv ntawm muaju. Qhov no provokes ib tug kab mob ntawm cov pob qij txha.

Gout yuav tshwm sim rau lwm yam yog vim li cas, xws li:

• cawv kom tsawg;
• lub cev ua ub no;
• kev poob plig;
• kab mob;
• los ntshav;
• tshuaj;
• radiotherapy.

Yuav ua li cas los tejzaum cov gout?

1. Muab tiav so. Thaum lub sij hawm exacerbation ntawm tus cov kev sib koom yuav tsum yog nyob rau hauv lub tsa txoj hauj lwm. Qhov no yuav tsis tau qhov mob.
2. Saib haus tsoom fwv. Nyob rau hnub yuav tsum haus dej haus tsawg kawg yog 5-6 tsom iav dej. Ib tug txaus tus nqi ntawm cov kua hauv lub cev nkoos qhov kev tshem tawm ntawm uric acid.
3. Tsis txhob siv tshuaj, nrog rau cov tshuaj aspirin, vim qhov no tshuaj slows acid.
4. Tshem tawm los ntawm high-protein noj cov zaub mov cov zaub mov nplua nuj nyob rau hauv purines, vim hais tias xws zaub mov tsub kom cov ntsiab lus ntawm uric acid nyob rau hauv lub cev.
5. Ib tug dhau heev lawm ntawm xws li ib tug yeeb tshuaj tau nyob rau hauv kev puas tsuaj rau cov pob qij txha, ob lub raum, cov hlab ntsha. Yog li ntawd, nws yuav tsum tau raws nrog kev kho mob muab rheumatologist.

kev tiv thaiv

Yog hais tias muaj yog ib tug kev tshuaj ntsuam genetic predisposition rau tus kab mob, nws yog tsim nyog los tsis tu ncua tswj tus nqi ntawm uric acid nyob rau hauv cov ntshav. Nyob rau hauv tas li ntawd, nws yog pom zoo kom ua ib tug noj qab nyob zoo txoj kev ua neej. Tus kab mob feem ntau muaj nyob rau hauv cov neeg uas nyhav dhau heev lawm. Healthy kev noj haus uas tsis muaj kev siv cov khoom uas muaj purines, yog ib feem ntawm tus kab mob tiv thaiv.

Yog hais tias koj muaj kev txhawj xeeb txog gout, dab tsi yog tus kab mob no thiab yuav ua li cas kho mob yog tsim nyog, qhia rau ib tug tej tus kws khomob tshwjxeeb. Tom qab tag nrho cov kws kho mob lub tswv yim pom zoo, koj yuav sai sai no pom tias tus kab mob no recedes.