Cerebral microangiopathy - dab tsi nws yog, ua, paub thiab kev kho mob

Angiopathy hlwb yog ib tug vascular kab mob, uas rau txawv yog vim li cas yog lawv cov laus, ua rau lub hlwb hlwb xav tsis nkag nrog cov ntshav. Qhov no yog ib tug heev loj pathology, uas yuav ua tau kom ntshav thiab tuag taus. Nyob rau hauv tej zaum peb yuav tham txog tej yam kab mob raws li cerebral microangiopathy. Yuav ua li cas yog nws? Nyob rau hauv cov ntaub ntawv no cuam tshuam me me hlab ntsha. Xav txog tus kab mob no nyob rau hauv ntau yam.

pathology yam ntxwv

Muaj coob tus tau hnov thiab ib txhia txawm ntsib nrog xws li cov kab mob li cerebral microangiopathy. Yuav ua li cas yog nws? Qhov no pathological txheej txheem uas muaj feem xyuam rau cov hlab ntsha thiab me me hlab ntsha uas acquires ntev. Nyob rau hauv lub paj hlwb, cov ntshav ncig yog disrupted. Txij li thaum rau tej ntshav txaus nyob rau hauv teb pa thiab cov zib, tus nruam tsis muaj cov tshuaj ua rau muaj kev cuam tshuam me me cov hlab ntsha ntawm lub paj hlwb.

ntaub ntawv microangiopathy

Qhov no lub hlwb pathology tau los ntawm kev cuam tshuam me me hlab ntsha. Qhov no yog manifested nyob rau hauv ib zaug, li ntawd, yuav kuaj tus kab mob no yog heev nyuaj nyob rau hauv thaum ntxov ua sawv.

Cerebral microangiopathy tshwm sim mas nyob rau hauv cov neeg laus. Ib tug tshwj xeeb uas muaj feem yuav pab pawg neeg kev txom nyem los ntawm tus kab mob Alzheimer. Nyob rau hauv xws li ib tug kab mob nyob rau hauv cov hlab ntsha phab ntsa pib noog amyloid (glycoprotein). Tsis tas li ntawd muaj nqis ntsoov teev yog hais tias lub pathology yog tsis zoo tus mob thiab kev kho mob.

Lentikulostriarnaya microangiopathy feem ntau muaj feem xyuam rau cov me nyuam thiab yog tsis yog ib tug kab mob, tab sis ib tug feature ntawm lub circulatory system. Yuav tsum muaj tas mus li xyuas.

Ntshav qab zib microangiopathy tshwm sim vim lub plawv mob nyob rau hauv lub hlwb uas ua ntshav qab zib.

Hypertensive microangiopathy tshwm sim los ntawm kev cuam tshuam ntawm cov ntshav system tsob vim tawg. Nws yog tsim nyog los tswj cov ntshav siab thiab tiv thaiv kom txhob surges.

Cov ua rau tus kab mob

Kev loj hlob ntawm microangiopathy tshwm sim tiv thaiv lwm yam kab mob. Qhov no tej zaum yuav muaj ntshav qab zib, mob cancer, siab pathology, hemolysis.

Ntawm cov ntsiab yog vim li cas yog cov nram qab no:

• tus kab mob (qhua pias, rubella);
• pathology pab mus rau lub yeej ntawm cov ntshav;
• mob ntshav qab zib;
• tawg;
• hnub nyoog laus;
• raws roj ntsha kab mob ua rau vascular laus;
• ntau yam kev raug mob;
• mob loj heev intoxication;
• kev tsim txom muaj yees.

Feem ntau cov feem ntau, ib tug pathology npaj raws li ib tug tshwm sim ntawm cov nram qab no dab:

• Necrosis - kev tuag ntawm ntaub so ntswg thiab hlwb.
• Hyalinosis - ib tug muaj tsis txaus cov protein nyob rau hauv lub keeb kwm yav dhau hyaline deposits.
• Nce permeability ntawm cov hlab ntsha. Raws li ib tug tshwm sim, lub fibers thiab cov yam ntxwv hais ntawm connective cov ntaub so ntswg yauv irreversible hloov.
• Thrombosis, tsiag ntawv los ntawm tsim ntawm ib tug ntshav los khov rau ntawd nyob rau hauv lub lumen ntawm cov hlab, uas ua rau cuam tshuam cov ntshav khiav.
• Kawg ntawm vascular endothelium, uas ua rau yus mus rau lawv spasm. Thaum xws li ib tug lub xeev yog txo cov khoom antithrombin, prostacyclin, thromboxane concentration yuav tsub kom.

Yam ntxwv nta microangiopathy

Soj ntsuam ces ntawm tus kab mob nyob ntawm seb cov kev hloov khoom nruab nrog thiab cov ntaub so ntswg kev puas tsuaj uas tshwm sim nyob rau hauv tus ntawm tej lwm yam. Cov neeg mob feem ntau yog tsis txaus siab ntawm kev mob thiab kub nov ntawm nqaij tawv nyob rau hauv lub ob txhais taw, tsis pom kev, los ntshav ntawm lub plab thiab lub qhov ntswg, qhov kev loj hlob ntawm sib quas ntus claudication, daim tawv nqaij dryness nres hemoptysis.

Yog li, muaj cov nram qab no nta microangiopathy:

• ua txhaum lub meej mom ntawm me me hlab ntsha;
• tsim lub raum tsis ua hauj lwm;
• puas ntshav liab;
• disturbed ntshav txheej txheem;
• qhov tshwm sim ntawm tshwm sim ntawm nqaij doog.

cov tsos mob

cerebral microangiopathy muaj cov nram qab cov tsos mob: tus neeg mob pib yws ntawm lub cim xeeb tsis meej, kiv taub hau, tsis muaj zog thiab muaj causeless mob taub hau. Lawv tshwm sim vim lub fact tias nyob rau hauv lub hlwb hlab ntsha yog ib tug ua txhaum ntawm cov ntshav ncig. Cov uas ua kev puas tsuaj rau cov phab ntsa hauv cov hlab ntsha me me activates oxidative kev ntawm lipids, dawb radicals yog tsim uas deleteriously cuam tshuam lub hlwb hlwb.

Raws li tus kab mob loj zuj zus tus neeg mob yuav inattentive thiab hnov qab. Nws zoo nkaus li staggering moj yam, chim siab convergence, tus menyuam kawm ntawv txoj kab uas hla yuav sib txawv, deteriorating nrig txog kev pom muaj nuj nqi. Nyob rau hauv tas li ntawd, muaj ntau zaus hauv qhov ntswg los ntshav thiab lub plab tsis xis nyob rau hauv lub ob txhais ceg ntawm so, thiab qhuav taw tshwv tawv nqaij, ntshav txhaws nyob rau hauv cov zis.

diagnostics

Yog hais tias muaj yog ib tug suspicion ntawm lub hlwb pathology, nws yog ib qho tseem ceeb rau kev tshawb nrhiav, qhov chaw uas lub hnab ris yuav nyob microangiopathy thiab dab tsi degree ntawm kev puas tsuaj rau cov hlab phab ntsa. Yuav kom qhov no kawg, lub nram qab no diagnostic txoj kev yog siv:

• MRI ntawm lub paj hlwb;
• nuclear magnetic resonance;
• Doppler ultrasound.

Yuav kom tau ntau npaum li cov lus qhia raws li tau siv ib tug kuj zoo txoj kev tib neeg lub cev, zoo li lub paj hlwb MRI, uas tus nqi ib tug me ntsis tom. Qhov no txoj kev ntawm mob yog tus nrov tshaj plaws. Nws tso cai rau kom paub tias tej yam tshwm sim ntawm tus kab mob xws li cerebral Atrophy, leykoareoz, perivascular tej qhov chaw expansion, me me hemorrhage, lacunar infarcts. MRI ntawm lub paj hlwb, tus nqi uas nyob rau ntawm tus nqi ntawm cov zoo nruab nrab siv, tso cai rau kom muab tso rau qhov tseeb kuaj mob. Yog hais tias tus neeg mob tsis nyhav tshaj 69 kg, daim duab yog 2,700 rubles, los ntawm 70 mus 99 kg - 3200 rubles, los ntawm 100 kg thiab ntau - 4400 rubles.

magnetic resonance imaging txoj kev tso cai rau txoj kev tshawb no ntawm lub paj hlwb cov ntaub so ntswg nws tus kheej thiab vasculature. Nyob rau hauv cov ntaub ntawv no, kom meej meej pom tej yam tshwm sim ntawm hypoplastic cov hlab ntsha. infarct yooj yim pom, uas tshwm sim vim vascular occlusion, cerebral hypoperfusion, me me los ntshav.

Doppler ultrasound txoj kev tso cai mus ntes hloov nyob rau hauv cov hlab ntsha, txhaws los yog narrowing.

kev kho mob

Cov neeg mob yeej tsis paub tias thaum twg lawv mob "cerebral microangiopathy", nws yog dab tsi thiab seb nws puas yuav tau kho. Tus kws kho mob feem ntau yog tsa ib tug complex txoj kev kho.

Ua ntej ntawm tag nrho cov, tus neeg mob yuav tsum noj tshuaj uas normalize cov ntshav siab. Nyob rau hauv thiaj li yuav tshem tawm cov tsis muaj oxygen nyob rau hauv lub hlwb, siv antihypoxants, xws li "Emoksipin". Nws tseem ceeb heev kom hloov cov lipid zoo siv statins, fibrates, kua tsib acid sequestrants. Qhov no tej zaum yuav muaj xws li cov nram qab no tshuaj: "Atorvastatin", "Simvastatin", "Rosuvastatin."

Tsis tas li ntawd, Yog hais tias koj paub hais tias microangiopathy, kev kho mob yuav tsum tau nqa tawm nrog rau kev siv nootropics uas txhim kho lub hlwb lub hwj chim. Qhov no "Piracetam," "Nootropil", "Tserebrozilin", "Encephabol." Yuav kom normalize lub plawv nicotinic acid npaj siv "Pikammilon", "Ksantilona nicotinate."

xaus

Yog li, peb tau sib tham txog tej yam kab mob raws li cerebral microangiopathy. Yuav ua li cas yog nws, tam sim no koj paub. Qhov no yog ib tug loj pathology uas yuav tsum tau tam sim ntawd kev pab. Tus hais tam sim no nws yog pom, qhov twg laus kom pom tseeb ntawm tus kab mob thiab cov neeg ntawm vascular txhab, qhov yooj yim dua nws yuav kom txo tau lub txim ntawm tus kab mob no.