Noj qab haus huvNpaj

Qabzib 5 feem pua: kev qhia, nyeem

Nyob rau hauv dab tsi ntau npaum yuav muab piam thaj 5 feem pua? Cov lus qhia ntawm no lub cuab tam yog piav rau hauv qab no. Tsis tas li ntawd, nws cov khoom uas hais hauv qab no, thiab cov hau kev ntawm kev siv indications.

Tus duab, ntim

Piam thaj 5 feem pua, cov kev qhia ntawv uas yog tau piav rau hauv qab no, nws muaj dextrose monohydrate. Qhov no Txoj kev lis ntshav npaj muag nyob rau hauv lub khob ntim, yas ntim, raws li zoo raws li nyob rau hauv polymer hnab, ntim rau hauv ib lub pob ntawm daim ntawv.

Lub zog ntawm cov tshuaj

Yuav ua li cas qab zib 5 feem pua? Phau ntawv hais tias qhov cuab tam yog muab kev koom tes nyob rau hauv metabolism hauv lub cev, thiab tej kev ncaj ncees thiab oxidative dab, pab txoj kev antitoxic siab thiab tsub kom lub contractile kev ua si ntawm lub plawv.

Peb tsis tau hais tias lub Txoj kev lis ntshav ntawm cov tshuaj no yog cov nyob H2O nyiaj tsawg. Tswv yim nyob rau hauv lub cev nqaij los, dextrose phosphorylated thiab hloov dua siab tshiab rau lub rau-glucose-phosphate, uas yog muaj nyob rau hauv lub ntsiab pauv units tib neeg lub cev tshuaj.

nta tshuaj

Lub zoo kawg li 5 feem pua zib? Phau Ntawv Qhia hais tias xws li ib tug tov muaj metabolic thiab detoxication txiav txim, thiab kuj sawv cev rau ib tug loj qhov chaw ntawm nkag assimilable thiab tseem ceeb as.

Nyob rau hauv tus txheej txheem ntawm cov metabolism hauv nyob rau hauv cov ntaub so ntswg ntawm dextrose tsim ib tug lossis loj npaum li ntawm lub zog, uas yog qhov tseem ceeb rau tej lub cev muaj nuj nqi.

Saib cov tshuaj yog isotonic. Nws lub zog tus nqi ntawm 200 kcal / l thiab ib tug approximate osmolarity - 278 mOsm / l.

kinetics

Yuav ua li cas yog qhov haum ntawm cov tshuaj, xws li zib 5 feem pua? Instruction (me nyuam yug tshiab txhais tau tias yog tus kws kho nkaus xwb los ntawm cov tshuaj) hais tias dextrose metabolism yog ntawm pyruvate thiab lactate mus rau cov dej carbon dioxide raws li los ntawm kev tso cai ntawm lub zog.

Cov tshuaj no yog kiag li absorbed, ob lub raum tsis yog tso zis (soj nyob rau hauv cov zis yog ib tug pathology).

Ntxiv pharmacokinetic thaj chaw ntawm cov tshuaj no yog txiav txim los ntawm txoj kev ntxiv rau nws.

Indications rau kev tswj hwm ntawm cov tshuaj

Vim li cas muaj peev xwm raug muab tso rau cov neeg mob zib 5 feem pua? Cov lus qhia (rau cov me nyuam thiab cov laus cov tshuaj no yog pom zoo kom mus thov rau tib lub indications) qhia tias qhov cuab tam yog siv nyob rau hauv cov nram no mob:

  • thaum extracellular isotonic lub cev qhuav dej;
  • raws li ib tug carbohydrate qhov chaw;
  • rau yug me nyuam thiab thauj cov tshuaj muab parenterally (i.e., raws li ib tug yooj yim tshuaj).

Bans cov kev taw qhia ntawm

Nyob rau hauv tej rooj plaub, cov neeg mob yuav tsis taw los ntawm lub 5 feem pua zib? Cov lus qhia (rau cov miv yog ib lub cuab tam yuav tsum pom zoo kom tsuas tej tug kws kho tsiaj) hais txog tej contraindications, xws li:

  • hyperosmolar coma ;
  • decompensated mob ntshav qab zib;
  • hyperglycemia;
  • impaired zib kam (xws li thaum lub sij hawm metabolic kev nyuaj siab);
  • giperlaktatsidemiya.

Ceev faj zib kws kho rau mob decompensated lub plawv tsis ua hauj lwm hom, hyponatremia, mob raum tsis ua hauj lwm (rau oliguria thiab anuria).

Piam thaj 5 feem pua: kev qhia

Rau cov dev thiab lwm yam tsiaj tshuaj no yog muab rau ib tug neeg hauv paus, nruj me ntsis rau cov lus tim khawv. Tib yam siv rau cov neeg.

Isotonic dextrose tshuaj yuav tsum tau muab intravenously ntawm ib tug nqi siab tshaj ntawm 150 ncos ib feeb. Cov pom zoo ntau npaum rau cov neeg laus cov neeg mob yog 500-3000 ml ib hnub twg.

Rau cov me nyuam mos thiab cov me nyuam mos uas mus txog 10 kg ntawm active medicament muab 100 mL / kg ib hnub twg. Tshaj cov qhia ntau npaum yog tsis pom zoo.

Cov neeg uas muaj mob ntshav qab zib mellitus dextrose yuav tsum tau nkag mus hauv tsuas nyob rau hauv cov kev tswj ntawm nws cov ntsiab lus nyob rau hauv cov zis thiab ntshav.

Cov pom zoo ntau npaum ntawm cov tshuaj thaum uas siv rau yug me nyuam thiab thauj ntawm parenterally muab tshuaj yog 50-250 ml ib 1 koob tshuaj muab neeg sawv cev.

phiv

Thaum siv kom pom tau hais tias tsis zoo li txhais tau tias kev phiv tshuaj. Nyob rau hauv tej rooj plaub, lub qab zib tov yog kev loj hlob ntawm hyperglycemia, kub taub hau, hypovolemia, mob laug ventricular tsis ua hauj lwm thiab polyuria.

Tej zaum koj kuj muaj kev hauv zos tshua xws li thrombophlebitis, txoj kev loj hlob ntawm tus kab mob, nqaij doog thiab lub zos ce.

tej ntaub ntawv

Nyob rau hauv chaw kho tsiaj xyaum, cov kev siv ntawm isotonic zib daws yog nyob rau hauv coob. Xws li ib tug kev npaj yog siv mus rau replenish lub active tsiaj lub cev dej thiab cov as-ham.

Raws li ib tug txoj cai, lub cuab tam yog muab rau miv, dev, yaj thiab lwm yam tsiaj uas muaj kua tsis, raug tshuaj lom, tshwm sim ntawm kev poob siab, lom, daim siab tus kab mob, hypotension, gastro-plab hnyuv kab mob, Aten acetonemia, gangrene, mob decompensation ua hauj lwm, hemoglobinuria thiab lwm lub xeev .

Depleted tsiaj yog suav tias yog tsis muaj zog thiab cov tshuaj raug raws li lub hwj chim ntawm cov kev npaj.

Ntau npaum thiab lawv cov koom haum saib xyuas

Tsiaj 5 feem pua zib tov twb muab intravenously los yog subcutaneously. Nyob rau tib lub sij hawm raws li cov nram qab no noj tshuaj npaum cas:

  • miv - 7-50 ml;
  • nees - 0,7-2,45 l;
  • dev - 0,04-0,55 l;
  • me me nyuj - 0,08-0,65 l;
  • npua - 0,3-0,65 l;
  • Nyuj - 0.5-3 l.

Los ntawm subcutaneous txhaj no koob tshuaj yog muab faib ua ob peb tshuaj tau nyob rau hauv txawv qhov chaw.

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