'De-Nol' - rau cov kev kho mob ntawm ulcers, erosions ntawm lub plab thiab duodenum

Kab mob ntawm lub plab nrog cev xws li ib tug loj pawg ntawm cov kab mob ntawm lub plab thiab cov hnyuv. Feem ntau cov kab mob yog mob gastritis thiab ulcers. Nyob rau hauv cov neeg mob, muaj ib tug tsis ua hauj lwm ntawm lub mechanism ntawm kev cai ntawm pais plab acid tsim, impaired piv ntawm acid-peptic ceeb thiab lub cev defenses, pais plab mucosal kev puas tsuaj nrog rau cov tsim ntawm ulcers thiab erosions.

Ua rau erosive gastritis thiab ulcers ntawm lub plab heev. Nyob rau hauv tsis ntev los no xyoo lawm, muaj ib tug ntau ntawm cov ntaub ntawv nyob rau hauv kev koom tes nyob rau hauv yuav ua mob rau sab hauv thiab txij nkawm ntawm o nyob rau hauv lub plab hauv ob sab phlu, gram-tsis zoo microorganism - Helicobacter pylori. Kev muaj ntawm cov kab mob tso cai rau nws rau koj tsiv nyob rau hauv ib tug khov nruab nrab xws li pais plab kua txiv. Nyob rau hauv gastritis thiab ulcers ntawm tus kab mob yog kuaj nyob rau hauv feem ntau. Nws muaj lub peev xwm yuav tsau thiab hauj lwm rau lub epithelial hlwb, hnoos qeev ntau lawm, thiab kom txhob muaj kev Process nws zoo.

Tej lub xeev mucosa txhawb equilibrium txhoj puab heev thiab defensive yam. Pais plab hnoos qeev yog ib tug muaj kev ruaj ntseg tivthaiv, tiv thaiv cov mucosa ntawm lub aggression ntawm hydrochloric acid. Muaj kuj yog tiv thaiv yam muaj xws li cov ntshav mov rau lub plab thiab ib tug zoo peev xwm los tsim dua tshiab. Pheev yam suav muaj zog zus ntawm hydrochloric acid thiab pepsin, cov neeg pluag kom ntawm lub plab, mob plab, poob plig tawv thiab duodenal Helicobacter pylori.

Tshuaj kev kho mob ntawm gastritis thiab pais plab rwj yog muab nruab nrab yog txoj gastroenterologist thiab cov tshuaj uas phiaj irradiation ntawm Helicobacter pylori, tsuj ntawm overproduction ntawm hydrochloric acid patronage mucosal lwm thiab erosions thiab ulcers.

Ib yam ntawm cov loj cov tshuaj siv nyob rau hauv ua ke nrog antiulcer txoj kev kho yog "De-Nol". Nyob rau hauv nws muaj pes tsawg leeg npaj "De-Nol" muaj Bismuth oxide. De-Nol thiab antiulcer kev ua si txuam nrog kev nyuaj siab loj (bactericidal) nyhuv on H. pylori kab mob. De-Nol yog tau envelop mucosa, txoj kev ua rau kev tiv thaiv sheath ntawm Bismuth tebchaw rau ulcerative thiab erosive tsis xws luag. Cov tshuaj muaj anti-inflammatory nyhuv, tsub kom cov kev ua ntawm kev tiv thaiv yam. Qhov no yog tiav los ntawm nce lub tsim ntawm prostaglandins, pais plab hnoos qeev thiab bicarbonate. Cov feem erosive los yog ulcerative defect De-Nol ua nce cov ntaub so ntswg lwm. Cov tshuaj cheeb qhov uas cov kev ua ntawm pepsin.

Bismuth tsis mus dhau hauv cov hnyuv, ib tug me me npaum li cas ntawm nws yog absorbed rau hauv cov ntshav. Lub tshuaj no yog muab tau los ntawm cov hnyuv.
Tshuaj "De-nol", siv nyob rau hauv cov kev kho mob ntawm ulcerative txhab ntawm lub plab thiab duodenum, nyob rau hauv mob, mob gastritis thiab gastroduodenitis, Gastroenteritis thiab dyspepsia.
Cov tshuaj no yog qhia rau cov kev kho mob ntawm cov kab mob nyob rau hauv cov neeg laus thiab cov me nyuam hluas li plaub xyoo. Muab xam los ntawm cov tshuaj ntau npaum nyob rau hauv cov me nyuam ua ib kilogram ntawm lub cev hnyav ntawm tus me nyuam. Nkag yog nqa tawm rau ib nrab ib teev ua ntej noj mov nrog ib tug me ntsis dej. Lub caij ntawm kev kho mob nyob rau qhov xwm ntawm tus kab mob no thiab nws yog feem ntau ib mus rau ob lub hlis.

Cov tshuaj no yog feem ntau zoo taus. Phiv tshua muaj tsawg tsawg thiab tej zaum yuav qhia nyob rau hauv dyspeptic phenomena thiab kev ua txhaum ntawm lub rooj zaum, thaum lub sij hawm mob thawj coj yuav tsim encephalopathy.

Cov tshuaj yuav tsum tsis txhob coj cov tsos mob raum tsis ua hauj lwm, cev xeeb tub thiab lactation.

Nyob rau hauv kev twb kev txuas nrog cov tseem ntawm tsub zuj zuj ntawm Bismuth noj cov tshuaj yuav tsum tsis txhob ntau tshaj lub sijhawm thiab ua ke nrog rau lwm cov Bismuth-muaj tshuaj.