Dermatitis kev kho mob

Nyob rau hauv xyoo tsis ntev los, cov neeg no nce tsis txaus siab hais txog cov tsos ntawm ntau hom ntawm dermatitis uas tshwm sim rau ib tug ntau yam yog vim li cas. By thiab loj, no muaj xws li yuav luag tag nrho cov inflammatory cov tshuaj tiv thaiv ntawm daim tawv nqaij nyob rau hauv raug rau ntau stimuli. Dermatitis kev kho mob pib tsuas yog tom qab qhov ua rau ntawm tus kab mob no yog kom meej meej tsim.

Dermatitis yog muab faib mus rau hauv kev sib cuag thiab tshuaj cov tshuaj tiv thaiv. Tiv tauj tshwm sim vim qhov tus ntawm lwm yam ntawm daim tawv nqaij. Thiab thaum toksidermii tej yam zoo ua ntej mus txeem mus rau hauv lub cev thiab ces manifest lawv tus kheej rau ntawm daim tawv nqaij. Pib kev kho mob ntawm dermatitis yog tsis yooj yim sua yam uas tsis tau paub nws etiology thiab pathogenesis. Tag nrho cov stimuli uas ua rau tus kab mob no, muaj ib tug tshuaj, lom los yog lub cev xwm. Qhov yooj yim txoj kev yog ua cov kev kho mob ntawm dermatitis tshwm sim los ntawm lub cev voos. Qhov no siab, kev sib txhuam, kub, thiab hluav taws xob kis, qhov kev txiav txim ntawm acids thiab alkalis. Feem ntau ua rau voos ntawm cov nroj tsuag xws li spurge, acrid buttercup, nettle, fraxinella, nyuj parsnip, thiab lwm tus neeg.

Lub thiaj li hu ua "yeem" irritants yuav ua rau o ntawm daim tawv nqaij nyob rau hauv tsuas yog cov neeg uas muaj ib tug muaj zog rhiab heev. Lawv ua tsis haum dermatitis. Nyob rau hauv feem ntau tus neeg mob, tus kab mob no ua rau cov nram qab no yam: formalin, Turpentine, tshuaj, polymers, detergents, perfumes, tshuaj pleev ib ce, tshuaj tua kab hauv lub ntsev ntawm npib tsib xee, cobalt, chromium. Nroj tsuag xws li paam dlev, haus luam yeeb, snowdrop, qij, geranium, primrose, kuj ua rau tus kab mob no.

Pathogenesis pa dermatitis txo xwb los puas ntawm daim tawv nqaij cov ntaub so ntswg, ces nws cov concentration txiav txim manifestation (lub zog), qhov xwm thiab ntev ntawm raug mus rau lub stimulus. Yog li daim tawv nqaij lesion tshwm sai li sai tau tom qab raug tus kab mob mus rau sab nraud stimuli, thiab nws cheeb tsam coincides nrog cov kev sib cuag cheeb tsam.

Sensitizers ua tsis haum dermatitis, txuas nrog daim tawv nqaij cov ntaub so ntswg proteins uas kom tsim conjugates uas muaj lub thaj chaw ntawm allergens. Nyob rau hauv tus ntawm lawv cov khoom yog tsa lymphocytes, uas leads mus rau kev loj hlob ntawm dermatitis, uas yog ib tug ncua hom kev tsis haum cov tshuaj tiv thaiv. Loj lub luag hauj lwm nyob rau hauv sensitizing mechanism plays tus neeg thaj chaw ntawm tus kab mob, namely tus mob ntawm poob siab system, kev tshuaj ntsuam genetic predisposition, ntawm daim tawv nqaij mob, thiab concomitant kev mob nkeeg thiab muaj nuj nqi ntawm hws thiab sebaceous qog. Monovalent sensitization txiav txim seb tus khiav cov yam ntxwv ntawm tej dermatitis: lub xub ntiag ntawm lub latent lub sij hawm, cov kev siv ntawm cov inflammatory tshua ntawm daim tawv nqaij, concentration ntawm cov stimulus lub sij hawm thiab nws cov teebmeem, cov puas cheeb tsam.

dermatitis kev kho mob yuav pib nrog ib tug txhais ntawm kev soj ntsuam kab mob. Tej yam yooj yim dermatitis tshwm sim nyob rau hauv mob los yog mob daim ntawv. Cov mob daim ntawv muaj 3 theem: erythematous (edema thiab hyperemia), vesicular (npuas tag, crusts thiab erosions), necrotic (decomposes cov ntaub so ntswg thiab tom ntej cirrhosis). Cov mob daim ntawv no yog los ntawm kev mob loj khaus, mob, mob nov ntawm nqaij tawv. Cov mob daim ntawv no, uas yog qhov ua rau ntawm lub caij nyoog raug mus rau lub stimuli, txawv txhaws erythema, lichenification, infiltration, ntes, enhanced keratinization, daim tawv nqaij Atrophy.

Dermatitis mob yog nqa tawm siv cov nram qab xeem: urinalysis thiab cov ntshav immunogram, quav helminth qe, fluorography, proteinogramma ntshiab. Raws li lawv muab kho tshuab, uas muaj: noj cov zaub mov; hyposensitization, sedatives, antihistamines; tshem tawm ntawm lub allergen; es rov ntawm lub cheeb tsam cov; txoj kev kho ntawm opportunistic kab mob; corticosteroids; Vitamins C, B; nicotinic acid; adaptogens; immunomodulatory txoj kev kho.

Tiv tauj Dermatitis kho mob yuav pib nrog lub ntsiab txhais ntawm lub stimulus thiab lub cessation ntawm kev sib cuag nrog nws. Therapy feem ntau yuav hydrocortisone tshuaj pleev, lwm yam steroid tshuaj pleev, siv ntawm ntub thiab txias compresses rau txhab.

Kev kho mob ntawm dermatitis ntawm lub ntsej muag yuav pib tsuas yog tom qab qhov ua rau ntawm tus kab mob no. Qhov no zoo ntawm dermatitis pib kho tom qab tshem tawm ntawm irritants (Tshuaj pleev ib ce, fluorinated tshuaj txhuam hniav, toning agents, intensive hnub raug, raug huab cua tej yam kev mob). Ua ib tug kev soj ntsuam ntawm cov neeg mob, cov kev kho mob yog nqa comorbidities endocrine thiab digestive systems. Nws yuav tsum tau ceev faj kev tu tawv nqaij. Nyob ntawm seb qhov ua rau, siv cov tshuaj thiab azelaic acid, txhais tau tias "Clindamycin", "Metronidazole". Zoo kev pab thiab txias gadgets nrog tannin tov thiab boric acid. Rau yav tom ntej pov thawj thiab sulfa tshuaj.